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Fig. 3 | BMC Infectious Diseases

Fig. 3

From: Antiviral mechanism of carvacrol on HSV-2 infectivity through inhibition of RIP3-mediated programmed cell necrosis pathway and ubiquitin-proteasome system in BSC-1 cells

Fig. 3

The molecular expression levels of each factor in cells treated with different post-infection time and different doses of carvacrol. a, b, c, d, and E represented mRNA levels (a. ICP4; b. ICP27; c.VP16; d. gB; e. UL30); f and g represented protein levels (f. the different HSV-2 post- infection time; g. the different concentration of carvacrol. *, p < 0.05; **, p < 0.01; ***, p < 0.001

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